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NHK is encoded by a nonsense mutation of α1-antitrpysin that results in a truncated product which is retained in the ER and undergoes ER-associated degradation, resulting in ER stress in diverse cell types (Nagasawa, Higashi, Hosokawa, Kaufman, & Nagata, 2007
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PDE5 inhibitors enhance tumor permeability and efficacy of chemotherapy in a rat brain tumor model