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Furthermore, the content of vitamin B1 in the plasma shows an inverse relationship with the level of glucose [29] A mutation in the methylenetetrahydrofolate reductase (MTHFR) gene or a homozygous TT genotype additionally impairs the metabolism of vitamins B9, B12, and B6, as well as the conversion of homocysteine (Hcy) to methionine, resulting in increased Hcy levels associated with inflammation [30]